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Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage


Journal article


J. Liu, S. Kasai, Y. Tatara, H. Yamazaki, J. Mimura, S. Mizuno, F. Sugiyama, S. Takahashi, T. Sato, T. Ozaki, K. Tanji, K. Wakabayashi, H. Maeda, H. Mizukami, Y. Shinkai, Y. Kumagai, H. Tomita, K. Itoh
International Journal of Molecular Sciences, vol. 23, 2022, p. 3201


https://www.mdpi.com/1422-0067/23/6/3201
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APA   Click to copy
Liu, J., Kasai, S., Tatara, Y., Yamazaki, H., Mimura, J., Mizuno, S., … Itoh, K. (2022). Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage. International Journal of Molecular Sciences, 23, 3201. https://doi.org/10.3390/ijms23063201


Chicago/Turabian   Click to copy
Liu, J., S. Kasai, Y. Tatara, H. Yamazaki, J. Mimura, S. Mizuno, F. Sugiyama, et al. “Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage.” International Journal of Molecular Sciences 23 (2022): 3201.


MLA   Click to copy
Liu, J., et al. “Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage.” International Journal of Molecular Sciences, vol. 23, 2022, p. 3201, doi:10.3390/ijms23063201.


BibTeX   Click to copy

@article{liu2022a,
  title = {Inducible Systemic Gcn1 Deletion in Mice Leads to Transient Body Weight Loss upon Tamoxifen Treatment Associated with Decrease of Fat and Liver Glycogen Storage},
  year = {2022},
  journal = {International Journal of Molecular Sciences},
  pages = {3201},
  volume = {23},
  doi = {10.3390/ijms23063201},
  author = {Liu, J. and Kasai, S. and Tatara, Y. and Yamazaki, H. and Mimura, J. and Mizuno, S. and Sugiyama, F. and Takahashi, S. and Sato, T. and Ozaki, T. and Tanji, K. and Wakabayashi, K. and Maeda, H. and Mizukami, H. and Shinkai, Y. and Kumagai, Y. and Tomita, H. and Itoh, K.}
}


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